The Classical Complement Cascade Mediates CNS Synapse Elimination

Therapeutic Targeting of the Complement System: From Rare Diseases to Pandemics

Complement and non-complement activating functions of C1q: A prototypical innate immune molecule - Annapurna Nayak, Lina Pednekar, Kenneth BM Reid, Uday Kishore, 2012

The Classical Complement Cascade Mediates CNS Synapse Elimination: Cell

Complement Protein C1q-Mediated Neuroprotection Is Correlated with Regulation of Neuronal Gene and MicroRNA Expression

Mechanisms governing activity-dependent synaptic pruning in the developing mammalian CNS

Local externalization of phosphatidylserine mediates developmental synaptic pruning by microglia

IJMS, Free Full-Text

C1q and the classical complement cascade in geographic atrophy secondary to age-related macular degeneration, International Journal of Retina and Vitreous

Emerging Roles of Complement Protein C1q in Neurodegeneration

The Classical Complement Cascade Mediates CNS Synapse Elimination: Cell

The Role of the Classical Complement Cascade in Synapse Loss During Development and Glaucoma

Complement C1q-dependent excitatory and inhibitory synapse elimination by astrocytes and microglia in Alzheimer's disease mouse models

Microglia and complement mediate early corticostriatal synapse loss and cognitive dysfunction in Huntington's disease

Complement cascade functions during brain development and neurodegeneration - Fatoba - 2022 - The FEBS Journal - Wiley Online Library

Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease