Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
Por um escritor misterioso
Last updated 28 setembro 2024
Chemokines encoded by herpesviruses - Pontejo - 2017 - Journal of Leukocyte Biology - Wiley Online Library
Frontiers Modulation of cellular signaling by herpesvirus-encoded G protein-coupled receptors
Innate metabolic responses against viral infections
The Chemokine Receptor D6 Constitutively Traffics to and from the Cell Surface to Internalize and Degrade Chemokines
International Union of Basic and Clinical Pharmacology. LXXXIX. Update on the Extended Family of Chemokine Receptors and Introducing a New Nomenclature for Atypical Chemokine Receptors
Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
Macrophage migration inhibitory factor is a valid drug target at the intersection of herpes simplex virus 1 replication and Alzheimer's disease-relevant cellular pathology
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner
Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
Role of neuroinflammation in neurodegeneration development
Chemokines and chemokine receptors in leukocyte trafficking American Journal of Physiology-Regulatory, Integrative and Comparative Physiology
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner
Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
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