Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
Por um escritor misterioso
Last updated 12 abril 2025


Chemokines encoded by herpesviruses - Pontejo - 2017 - Journal of Leukocyte Biology - Wiley Online Library

Frontiers Modulation of cellular signaling by herpesvirus-encoded G protein-coupled receptors

Innate metabolic responses against viral infections

The Chemokine Receptor D6 Constitutively Traffics to and from the Cell Surface to Internalize and Degrade Chemokines

International Union of Basic and Clinical Pharmacology. LXXXIX. Update on the Extended Family of Chemokine Receptors and Introducing a New Nomenclature for Atypical Chemokine Receptors

Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization

Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization

Macrophage migration inhibitory factor is a valid drug target at the intersection of herpes simplex virus 1 replication and Alzheimer's disease-relevant cellular pathology

Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner

Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization

Role of neuroinflammation in neurodegeneration development

Chemokines and chemokine receptors in leukocyte trafficking American Journal of Physiology-Regulatory, Integrative and Comparative Physiology

Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner

Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization

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